An obstruction in the aqueous humor’s outflow between the anterior and posterior chambers of the eye causes acute angle closure, an emergency ophthalmic disorder that, if untreated, can cause secondary visual neuropathy and an abrupt increase in intraocular pressure. The most frequent primary cause in patients with pre-existing narrow angles, such as those who are long-sighted, is a pupillary block. The usage of frequently prescribed drugs is one such secondary cause of angle closure that has to be made more widely known. To rule out other potential confounding illnesses, particularly those with CNS origins, that appear similarly, a thorough questioning is necessary.(1) All patients presenting with abrupt onset of red eye along with pupillary dilatation, dull pain, and headache should have angle closure excluded. A fundamental eye exam should involve a thorough neurological examination, measurement of intraocular pressure, and evaluation of the anterior segment under a strong light. When there is a strong clinical suspicion of acute angle closure, systemic ocular hypotensive therapy must be administered as away in order to protect the optic nerve and reduce vision loss. To specify definite care, an immediate referral to an ophthalmologist is required (2).